Contact:

At RegeneRx:
J.J. Finkelstein
jjfnk@regenerx.com
301.280.1992

At the Financial
Relations Board:
Analyst Inquiries:
Leslie Loyet
312.640.6672

or
General Inquiries
Kathy Waller
312.640.6696

or
Media Inquiries
Tim Grace
312.640.6741

RegeneRx Biopharmaceuticals, Inc. (OTCBB:RGRX) (www.regenerx.com) today announced an interview with CEO, J.J. Finkelstein, regarding recent milestones and current product development strategy. The interview is posted on the Wall Street Reporter Web site, www.wallstreetreporter.com.

"We're please to have the opportunity to discuss some of our plans for 2005 at a time when we are conducting Phase II human clinical trials with our wound healing drug, Thymosin Beta 4 (Tß4). Given the unique capabilities of Tß4, the success of research conducted at the NIH, the recent cardiac work at UT Southwestern Medical School, along with research conducted at other institutions, we anticipate an exciting year ahead," said Mr. Finkelstein.

About RegeneRx Biopharmaceuticals, Inc.
RegeneRx is a biopharmaceutical company developing Tß4 as a platform technology for the treatment of acute and chronic wounds and for the treatment of a variety of human diseases involving tissue and organ repair under an exclusive world-wide license from the National Institutes of Health. The technology is also the subject of over forty-five world-wide patents and patent applications held by RegeneRx related to dermal, ocular, and internal wounds and tissue repair, including the treatment and prevention of cardiac injury. RegeneRx recently received clearance from the U.S. FDA to conduct the first in a series of Phase II dermal wound healing clinical trials with Tß4.

Thymosin Beta 4
Tß4 is a naturally occurring peptide present in virtually all human cells. It represents a new class of wound healing drug and is different from other wound repair factors, such as growth factors, in that it promotes endothelial cell differentiation and keratinocyte cell migration, and down-regulates a number of inflammatory cytokines and chemokines. A key mechanism of action is Tß4's ability to regulate the cell-building protein, actin, a vital component of the cytoskeleton. Additionally, it has recently been reported that Tß4 directly influences the production of laminin-5, a protein necessary for proper adhesion of certain types of mammalian cells and an important component of the wound healing process. It has also recently been reported that Tß4 can inhibit or prevent apoptosis (programmed cell death) in ocular and cardiac tissues. Researchers at the National Institutes of Health (NIH), and at other academic institutions throughout the U.S., have published numerous scientific articles indicating that Tß4 is effective in accelerating dermal and corneal wounds in several animal models, under a variety of conditions.

Safe Harbor Statement
The information in this press release may include certain forward-looking statements within the meaning of the Private Securities Litigation Reform Act of 1995. These statements concern the Company's current expectations regarding future events, including the ongoing and prospective development of Tß4 and possible future benefits to the Company, its stockholders, and patients. Due to the nature of product development and the regulatory approval process, the forward-looking statements are subject to risks and uncertainties, including those reflected in the Company's filings with the Securities and Exchange Commission, particularly its most recent Annual Report on Form 10-KSB. The Company assumes no obligation to update or revise any forward-looking statements made herein or any other forward-looking statements made by the Company.

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